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Why is this post here in discussion? It is an example of how discussion proceeds in an area that is actually public health, ostensibly diet and health in the context of inflammation mediated disease processes caused by diet, specifically processed foods. We often have discussions that work like this one seems to be going: people with strongly rooted and divergent beliefs. But in terms of "position" it is very clear why those differences are likely to clash.
Here are two points of view at a level anyone here can easily see and understand:
A: https://www.foodnavigator-usa.com/A...weeteners-Association-Calorie-Control-Council
B: Lustig R., "Metabolical", pp 178-81 Artificial sweeteners section
Discussion point: "diet sweetener consumption is also correlated with metabolic disease, very like sucrose".
I think both sides agree on this part:
Metabolic disease is a collection of diseases that are caused by inflammation. One example is insulin resistance:
https://www.nature.com/articles/s41598-021-86993-7
Think of it as excessive insulin secretion as a positive feedback biochemical loop.
You can probably guess a priori which contributor wants to represent "pro" and which one "con".
Remember "correlation is not causation". So both sides can posit some credibility. As a guess. The discussion is really about insulin resistance caused by consuming processed foods with sweeteners. Sucrose (table sugar, a dimer of fructose and glucose) is a reasonably well established causative factor, so the logical point would be are sweeteners okay to replace sucrose? Do sweeteners cause an insulin misfire by the pancreas?
Here are two points of view at a level anyone here can easily see and understand:
A: https://www.foodnavigator-usa.com/A...weeteners-Association-Calorie-Control-Council
B: Lustig R., "Metabolical", pp 178-81 Artificial sweeteners section
Discussion point: "diet sweetener consumption is also correlated with metabolic disease, very like sucrose".
I think both sides agree on this part:
Metabolic disease is a collection of diseases that are caused by inflammation. One example is insulin resistance:
https://www.nature.com/articles/s41598-021-86993-7
Think of it as excessive insulin secretion as a positive feedback biochemical loop.
You can probably guess a priori which contributor wants to represent "pro" and which one "con".
Remember "correlation is not causation". So both sides can posit some credibility. As a guess. The discussion is really about insulin resistance caused by consuming processed foods with sweeteners. Sucrose (table sugar, a dimer of fructose and glucose) is a reasonably well established causative factor, so the logical point would be are sweeteners okay to replace sucrose? Do sweeteners cause an insulin misfire by the pancreas?