Do Rods play a role in daytime vision?

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Rod photo-sensitive cells do contribute to daytime vision, primarily by detecting overall light levels rather than fine details. They are adept at sensing large, homogenous shapes and slow-moving objects, which can be beneficial in various animals, not just humans. In mammals, rods are mainly located in the peripheral retina, where visual resolution is lower compared to the fovea, which contains a higher concentration of cones. Conditions like rod monochromacy and complete achromatopsia highlight the limitations of rods in bright light, as these patients experience photophobia and reduced visual acuity. Rods take time to adapt when transitioning from bright to dim environments, affecting their light-sensing capabilities. Additionally, rod photoreceptors are sensitive to light but produce neural signals more slowly than cones, which impacts visual perception in varying light conditions. High illumination can also damage rod cells, further complicating their role in vision.
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Do Rod photo-sensitive cells contribute at all to daytime vision?

If so, what exactly do they contribute? For example, they contribute towards big homogenous shapes, slow moving big objects, etc. I'm interested in whether this applies to any animal not just humans.
 
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I don't think there is any theory as to how nerve signals from optic sensors lead to perceptions of the world.
Other than that more light perception generally makes creatures more excited.
That applies to plants as well.
As far as I know, the rod sensors in mammals are good at sensing overall level of light.
This regardless of wavelength or time of day.
 
I think the best way to answer this question is to look for information on diseases which block cone cell activity but not rod cell activity. Rare genetic diseases will do this. Such patients have photophobia because rods don't work well in bright light. I think visual acuity increases as light dims. I found this reference describing patients with this condition, variously called rod monochromacy, complete achromatopsia or hemeralopia/day-blindness:

"In typical complete achromatopsia, patients present usually by 6 months old with photophobia and nystagmus. Visual acuity is typically less than 20/200 for those with complete achromatopsia... Photosensitivity persists and may remain a debilitating symptom."
Remmer MH, Rastogi N, Ranka MP, Ceisler EJ. Achromatopsia: a review.
 
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In humans, rods are predominately located in the periphery of the retina, which are areas of reduced resolution (this would be like areas of peripheral vision. The fovea (central point of the retina which is normally directed to points of interest) has both the highest concentration of photoreceptors and the most cones.
If the cones are replaced by rods then the rods may be able to make up some of the lost resolution @Chatul discussed (different diseases causes could do this differently), if not their retinal distribution might help explain those results.

When one goes from a highly illuminated environment to a dimly lit one, it takes several minutes for the rod cells to adapt to the darker levels of illumination. Until that happens, the rods are not that effective at sensing light. This is well know to those who have to have good night vision (for example those who are watch on ships at night).

In addition, rod photoreceptor molecules, rodopsin are located in disks of the outer segment (see wikipedia picture). The disks have a lifespan and are periodically shed and replaced. This happens more quickly at higher levels of illumination and could affect you scenario.
While rod photoreceptors are much omre sensitive to low light levels, an apparent tradeoff is that they are slower to produce a neural signal in response to light. This would also affect the no cone scenario.

It appears that there are also circumstances where high illumination can kill rod cells.
 
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